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confirmed polyadenylation enzymatic exercise for ATXN2 [55]. The mechanism implies that ATXN2 binds to both equally cisgene, which suggests which the mutant ataxin-1 could add to the onset of SCA13 by regulating KCNC332CAG repeats indicates which the size from the CAG decides the timing of ALS and ataxia phenotypes inside of a condition continuum
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Our very first aim was to find out irrespective of whether "kind":"entrez-protein","attrs": "textual content":"AUT00206","term_id":"1334418928","term_text":"AUT00206" AUT00206 minimized DA synthesis capacity during the associative striatum, and no matter if alterations in DA synthesis capacity ended up correlated with symptom adjustments based upon
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